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<td><span style="font-family:Helvetica, sans-serif; font-size:20px;font-weight:bold;">Mind & Brain News -- ScienceDaily</span></td>
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<td><a href="https://www.sciencedaily.com/releases/2026/03/260307213234.htm" style="font-family:Helvetica, sans-serif; letter-spacing:-1px;margin:0;padding:0 0 2px;font-weight: bold;font-size: 19px;line-height: 20px;color:#222;">Brain scans reveal how ketamine quickly lifts severe depression</a>
<div style="font-family:Helvetica, sans-serif; text-align:left;color:#999;font-size:11px;font-weight:bold;line-height:15px;">Mar 8th 2026, 19:40</div>
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<p>A new brain-imaging study has revealed how ketamine produces its fast antidepressant effects in people with treatment-resistant depression. Researchers tracked changes in a critical brain receptor that helps neurons communicate and found that ketamine reshapes its activity in specific brain regions tied to mood and reward. These shifts strongly matched improvements in patients’ symptoms. The findings could help scientists develop better ways to predict who will benefit from ketamine therapy.</p>
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<td><a href="https://www.sciencedaily.com/releases/2026/03/260307213244.htm" style="font-family:Helvetica, sans-serif; letter-spacing:-1px;margin:0;padding:0 0 2px;font-weight: bold;font-size: 19px;line-height: 20px;color:#222;">165,000 dementia patients reveal hidden stroke risk from common drug</a>
<div style="font-family:Helvetica, sans-serif; text-align:left;color:#999;font-size:11px;font-weight:bold;line-height:15px;">Mar 8th 2026, 16:45</div>
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<p>A massive UK study of more than 165,000 dementia patients has uncovered troubling new evidence about risperidone, a commonly used drug for severe agitation. Researchers found the antipsychotic increases stroke risk in dementia patients across the board—including those with no prior heart disease or stroke—challenging the long-held belief that some patients might be safer candidates than others. Because risperidone is the only drug of its type licensed for dementia, doctors often use it as a last resort when distressing symptoms become unmanageable.</p>
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<td><a href="https://www.sciencedaily.com/releases/2026/03/260307213232.htm" style="font-family:Helvetica, sans-serif; letter-spacing:-1px;margin:0;padding:0 0 2px;font-weight: bold;font-size: 19px;line-height: 20px;color:#222;">A new “magic mushroom” drug could treat depression without psychedelic hallucinations</a>
<div style="font-family:Helvetica, sans-serif; text-align:left;color:#999;font-size:11px;font-weight:bold;line-height:15px;">Mar 8th 2026, 06:26</div>
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<p>Scientists are exploring a new way to harness the medical promise of psychedelic compounds without the mind-bending side effects. Researchers created modified versions of psilocin — the active form of psilocybin from “magic mushrooms” — that still target key serotonin pathways linked to depression and other brain disorders but appear to cause far fewer psychedelic-like effects.</p>
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<td><a href="https://www.sciencedaily.com/releases/2026/03/260307155945.htm" style="font-family:Helvetica, sans-serif; letter-spacing:-1px;margin:0;padding:0 0 2px;font-weight: bold;font-size: 19px;line-height: 20px;color:#222;">Scientists discover hidden brain cells that may stop Alzheimer’s tau buildup</a>
<div style="font-family:Helvetica, sans-serif; text-align:left;color:#999;font-size:11px;font-weight:bold;line-height:15px;">Mar 8th 2026, 06:14</div>
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<p>Scientists have uncovered a surprising new role for little-known brain cells called tanycytes that may influence the development of Alzheimer’s disease. These specialized cells appear to help remove toxic tau protein from the brain by transporting it from the cerebrospinal fluid into the bloodstream. When tanycytes become damaged or dysfunctional, tau can accumulate in the brain, a hallmark of Alzheimer’s.</p>
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<p><strong>Forwarded by:<br />
Michael Reeder LCPC<br />
Baltimore, MD</strong></p>
<p><strong>This information is taken from free public RSS feeds published by each organization for the purpose of public distribution. Readers are linked back to the article content on each organization's website. This email is an unaffiliated unofficial redistribution of this freely provided content from the publishers. </strong></p>
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