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<td><span style="font-family:Helvetica, sans-serif; font-size:20px;font-weight:bold;">Mind & Brain News -- ScienceDaily</span></td>
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<td><a href="https://www.sciencedaily.com/releases/2026/01/260115022811.htm" style="font-family:Helvetica, sans-serif; letter-spacing:-1px;margin:0;padding:0 0 2px;font-weight: bold;font-size: 19px;line-height: 20px;color:#222;">One protein may decide whether brain chemistry heals or harms</a>
<div style="font-family:Helvetica, sans-serif; text-align:left;color:#999;font-size:11px;font-weight:bold;line-height:15px;">Jan 15th 2026, 23:01</div>
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<p>Tryptophan does far more than help us sleep—it fuels brain chemistry, energy production, and mood-regulating neurotransmitters. But as the brain ages or develops neurological disease, this delicate system goes awry, pushing tryptophan toward harmful byproducts linked to memory loss, mood changes, and sleep problems.</p>
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<td><a href="https://www.sciencedaily.com/releases/2026/01/260115022756.htm" style="font-family:Helvetica, sans-serif; letter-spacing:-1px;margin:0;padding:0 0 2px;font-weight: bold;font-size: 19px;line-height: 20px;color:#222;">This after-meal blood sugar spike may raise Alzheimer’s risk</a>
<div style="font-family:Helvetica, sans-serif; text-align:left;color:#999;font-size:11px;font-weight:bold;line-height:15px;">Jan 15th 2026, 07:51</div>
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<p>Spikes in blood sugar after eating may be more dangerous for the brain than previously thought. In a massive genetic study, people with higher post-meal blood sugar had a much greater risk of Alzheimer’s disease. The effect couldn’t be explained by visible brain damage, suggesting hidden biological pathways may be involved. Researchers say managing blood sugar after meals could become a key strategy for reducing dementia risk.</p>
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<p><strong>Forwarded by:<br />
Michael Reeder LCPC<br />
Baltimore, MD</strong></p>
<p><strong>This information is taken from free public RSS feeds published by each organization for the purpose of public distribution. Readers are linked back to the article content on each organization's website. This email is an unaffiliated unofficial redistribution of this freely provided content from the publishers. </strong></p>
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