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<td><span style="font-family:Helvetica, sans-serif; font-size:20px;font-weight:bold;">Mind & Brain News -- ScienceDaily</span></td>
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<td><a href="https://www.sciencedaily.com/releases/2025/11/251128050509.htm" style="font-family:Helvetica, sans-serif; letter-spacing:-1px;margin:0;padding:0 0 2px;font-weight: bold;font-size: 19px;line-height: 20px;color:#222;">Scientists uncover the brain’s hidden learning blocks</a>
<div style="font-family:Helvetica, sans-serif; text-align:left;color:#999;font-size:11px;font-weight:bold;line-height:15px;">Nov 28th 2025, 09:09</div>
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<p>Princeton researchers found that the brain excels at learning because it reuses modular “cognitive blocks” across many tasks. Monkeys switching between visual categorization challenges revealed that the prefrontal cortex assembles these blocks like Legos to create new behaviors. This flexibility explains why humans learn quickly while AI models often forget old skills. The insights may help build better AI and new clinical treatments for impaired cognitive adaptability.</p>
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<td><a href="https://www.sciencedaily.com/releases/2025/11/251126095034.htm" style="font-family:Helvetica, sans-serif; letter-spacing:-1px;margin:0;padding:0 0 2px;font-weight: bold;font-size: 19px;line-height: 20px;color:#222;">Hidden mitochondrial DNA damage may be a missing link in disease</a>
<div style="font-family:Helvetica, sans-serif; text-align:left;color:#999;font-size:11px;font-weight:bold;line-height:15px;">Nov 28th 2025, 04:29</div>
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<p>Researchers identified a new, sticky form of mitochondrial DNA damage that builds up at dramatically higher levels than in nuclear DNA. These lesions disrupt energy production and activate stress-response pathways. Simulations show the damage makes mtDNA more rigid, possibly marking it for removal. The finding offers fresh clues to inflammation, aging, and diseases such as diabetes and neurodegeneration.</p>
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<p><strong>Forwarded by:<br />
Michael Reeder LCPC<br />
Baltimore, MD</strong></p>
<p><strong>This information is taken from free public RSS feeds published by each organization for the purpose of public distribution. Readers are linked back to the article content on each organization's website. This email is an unaffiliated unofficial redistribution of this freely provided content from the publishers. </strong></p>
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